C. reinhardtii is a valid model for motility defects in SPGF — ARCADIA Knowledge Graph

Association·arcadia

C. reinhardtii is a valid model for motility defects in SPGF

Claim that despite baseline motility regulation differences, C. reinhardtii models motility defects relevant to spermatogenic failure disorders

Confidence
90%
active

Evidence Quote

“These results support C. reinhardtii as a model for studying motility defects despite baseline regulation differences”

Relationship

Chlamydomonas reinhardtii motility models Spermatogenic failure 43 (SPGF43)

Arguments

Chlamydomonas reinhardtii motilitysubject
Spermatogenic failure 43 (SPGF43)object

Connections (2)

Drug-specific modulation of motility in Chlamydomonas mutantsInferenceChain
Vesicle trafficking and glycosylation are required for notochord lumen inflationAssociation

Evidence

“Evidence describing NIH support for model organism research, associated with the cited reference.”

A Look at NIH Support for Model Organisms, Part Two

“Evidence summarizing publication trends in research involving model organisms, associated with the cited reference.”

Publication Trends in Model Organism Research

“Evidence summarizing mouse models of human disease from an evolutionary perspective, linked to the cited reference.”

Mouse Models of Human Disease: An Evolutionary Perspective